SAPS

SLEEP APNEA PREGNANCY SYNDROME

 

Medical Literature Review

 

By Charles L. McPhee, R.PSG.T.

mcphee@dreamdoctor.com

 

 

 

 

Am Rev Respir Dis. 1991 Aug;144(2):461-3.

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Obstructive sleep apnea during pregnancy. Therapy and implications for fetal health.

Charbonneau M, Falcone T, Cosio MG, Levy RD.

Desmond N. Stoker Laboratory,
Royal Victoria Hospital, McGill University, Montreal, Quebec, Canada.

A 32 yr-old woman in her last trimester of pregnancy was found to have severe obstructive sleep apnea (OSA). The overnight polysomnogram demonstrated an apnea plus hypopnea index of 159 events per hour. Apneas were associated with severe oxygen desaturation to 40% during rapid eye movement sleep, maternal bradycardia, and second degree heart block. External cardiotocography showed normal fetal heart rate reactivity to fetal movements, even during the apneas and episodes of oxygen desaturation. Nasal continuous positive airway pressure at a level of 15 cm H2O effectively treated the apneas and desaturation and had no effect on the fetal heart rate. The patient was induced electively during the 39th wk of pregnancy and gave birth to a newborn with growth retardation. Early recognition and treatment of OSA in pregnancy might prevent problems with fetal development.


 

Obstet Gynecol Surv. 1996 Aug;51(8):503-6.

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Obstructive sleep apnea in pregnancy.

Lefcourt LA, Rodis JF.

Department of Obstetrics and Gynecology, University of
Connecticut Health Center Farmington 06030-2950, USA.

Obstructive sleep apnea is a common medical condition characterized by periodic apneas during sleep that produce hypoxia and sleep disruption. Several cases of obstructive sleep apnea in pregnancy have been reported. In the case reported the woman admitted for evaluation and management of preeclampsia was observed experiencing apneic episodes and oxygen desaturation during sleep. Subsequent evaluation confirmed the diagnosis of obstructive sleep apnea. Pregnancy may be complicated by obstructive sleep apnea with potential adverse effects on the mother and fetus. Prompt diagnosis would allow early treatment and may prevent low birth weight and the development of preeclampsia.


 

J Am Board Fam Pract. 2004 Jul-Aug;17(4):292-4.

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Obstructive sleep apnea in pregnancy.

Roush SF, Bell L.

Cox Family Practice Residency,
Springfield, Missouri, 65802, USA. scott.roush@coxhealth.com

A 25-year-old woman, gravida 4 para 2, at 37 weeks gestation was evaluated and treated for preeclampsia. Overnight, the patient had a witnessed apneic episode with maternal oxygen desaturation and concurrent fetal heart rate deceleration. She subsequently delivered an infant that was small for gestational age. This is the first case described with confirmed obstructive sleep apnea by formal polysomnography and witnessed maternal desaturation with fetal heart rate decelerations. Recognizing obstructive sleep apnea (OSA) early in gestation will help dictate treatment options and may prevent adverse maternal fetal outcomes. Continuous positive airway pressure (CPAP) seems to be a safe treatment with minimal adverse effects. Questioning of patients at the first prenatal visit and monitoring for increased snoring during gestation may help detect early signs and symptoms of OSA. Treatment of OSA with CPAP might improve perinatal outcomes.

 

 

 

Eur J Obstet Gynecol Reprod Biol. 1978 Apr;8(2):77-81.

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Fetal response to periodic sleep apnea: a new syndrome in obstetrics.

Joel-Cohen SJ, Schoenfeld A.

Periodic sleep apnea, a chronic sleep deprivation state, in which marked changes in the arterial PO2 and PCO2 tensions have been recorded, is a relatively new syndrome not previously reported in pregnancy. It is characterized by episodes of apnea, prevalently obstructive, during sleep. The majority of patients with this syndrome have snored heavily for years, suggesting a causal relationship between snoring and periodic sleep apnea. The effects of prolonged snoring on alveolar ventilation and systemic pressure(s) suggest that this snoring has physiopathological implications on maternal cardio-respiratory reserve and indirectly upon the fetus, especially as there are recordable changes in fetal heart rate and also a change in the acid-base status of the fetus. The possibility that this syndrome may have an adverse effect upon the fetus is stressed.


 

Lin Chuang Er Bi Yan Hou Ke Za Zhi. 2002 Jun;16(6):295-6.

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[Obstructive sleep apnea of gestational period]

[Article in Chinese]

Zhu W, Shu C.

Wuxi Hospital for Maternal and Child Health Care, Wuxi 214062.

OBJECTIVE: To explore the influence of obstructive sleep apnea syndrome (OSAS) on the pregnant women. METHOD: 465 women in pregnancy were studied by random. The snoring rate, the relation with hyper blood pressure and preeclamptism, and the influence on infant were observed. RESULT: The magnificent statistics shows total snoring rate is 24.7%(115/465). 21.3%(92/442) is after pregnancy, of them, 28.7% meet the standard of OSAS, 12.17% with hyper blood pressure, 7.8% with preeclamptism, 5.2% infant with aplasia. CONCLUSION: Diseases incidence with OSAS is more obvious than that with normal pregnant women. OSAS has a marked influence on pregnancy. Pregnant women with obvious OSAS can be regard as an independent factor of high risk pregnancy. We should pay more attentions to them in their early stage.

 

 

 

Clin Chest Med. 1992 Dec;13(4):637-44.

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Respiration during sleep in pregnancy.

Feinsilver SH, Hertz G.

State University of New York, Stony Brook.

Several changes in maternal physiology may profoundly alter sleep, especially during late pregnancy. Any condition that causes maternal hypoxemia will be worsened during sleep, particularly in the supine position. Although high circulating levels of progesterone increase respiratory drive during sleep, in at least some women this protective mechanism is insufficient to prevent sleep-disordered breathing and hypoxemia. The true incidence of sleep-disordered breathing during pregnancy remains unknown. Although many women report sleep disturbance during pregnancy, those with severe snoring, observed irregular breathing with sleep, or excessive daytime somnolence should be referred for clinical polysomnography. With few data thus far available, nasal CPAP would appear to be the treatment of choice. Given the possible consequences of sleep apnea for fetal outcome, any significant sleep-disordered breathing is probably an indication for treatment.



 

Thorax. 2002 Jun;57(6):555-8.

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Sleep disordered breathing and pregnancy.

Edwards N, Middleton PG, Blyton DM, Sullivan CE.

David Read Laboratory, Department of Medicine,
University of Sydney, NSW 2006, Australia.

Many changes in the respiratory system occur during pregnancy, particularly during the third trimester, which can alter respiratory function during sleep, increasing the incidence and severity of sleep disordered breathing. These changes include increased ventilatory drive and metabolic rate, reduced functional residual capacity and residual volume, increased alveolar-arterial oxygen gradients, and changes in upper airway patency. The clinical importance of these changes is indicated by the increased incidence of snoring during pregnancy, which is likely also to reflect an increased incidence of obstructive sleep apnoea/hypopnoea syndrome. For the respiratory physician asked to review a pregnant patient, the possibility of sleep disordered breathing should always be considered. This review first examines the normal physiological changes of pregnancy and their relationship to sleep disordered breathing, and then summarises the current knowledge of sleep disordered breathing in pregnancy.

 

 

Curr Opin Pulm Med. 2000 Nov;6(6):485-9.

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Complications and consequences of obstructive sleep apnea.

Harding SM.

University of Alabama at Birmingham Sleep-Wake Disorders Center, Department of Medicine, University of Alabama at Birmingham, 35294, USA. sharding@uab.edu

Obstructive sleep apnea (OSA) has many consequences. There is an independent association between OSA and hypertension. The Sleep Heart Health Study reported that hypertension prevalence increased as sleep disordered breathing severity increased. The Nurses' Health Study noted an age-adjusted relative risk of cardiovascular events of 1.46 for occasional snorers and 2.02 for regular snorers, and a risk of stroke of 1.60 for occasional snorers and 1.88 for regular snorers. Sleep apnea is also associated with pulmonary hypertension, neurocognitive effects, depressed quality of life, motor vehicle accidents, awakening headache, childhood growth interruption, pregnancy-induced hypertension, fetal growth retardation, and disruption of the patients' bed-partners' sleep quality. Further research will examine the possibility of causality, pathophysiologic mechanisms, and outcomes of therapeutic interventions for OSA on the many consequences of OSA.

 

 

Curr Opin Pulm Med. 2003 Nov;9(6):477-83.

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Sleep disorders in pregnancy.

Sahota PK, Jain SS, Dhand R.

Department of Neurology and University of Missouri-Sleep Disorders Center, University of Missouri Health Care, Columbia, Missouri 65212, USA. sahotap@health.missouri.edu

PURPOSE OF REVIEW: Sleep disturbances are frequent during pregnancy. The spectrum of association between pregnancy and sleep disturbances ranges from an increased incidence of insomnia, nocturnal awakenings, and parasomnias (especially restless legs syndrome) to snoring and excessive sleepiness. These disturbances occur as a result of physiologic, hormonal, and physical changes associated with pregnancy. Although the timing and occurrence of different sleep disorders varies, they are most prevalent during the third trimester. Despite reports of the various sleep problems, the exact nature and incidence of sleep disorders in pregnancy is not known. Given that limitation, we are presenting an up-to-date review of the current understanding of the relation between sleep and pregnancy. RECENT FINDINGS: Studies suggest that pregnancy affects sleep in multiple ways. There are hormonal changes, physiologic changes, physical factors, and behavioral changes in a pregnant woman-all of which may affect her sleep. They may affect the duration and quality of sleep and lead to a variety of sleep disorders. Pregnancy may also affect an existing sleep disorder. Particular attention may be given to obese pregnant women who would gain more weight during pregnancy or those who develop hypertensive conditions (eg, preeclampsia). Snoring may be more common in women with preeclampsia and the pressor responses to obstructive respiratory events during sleep may be enhanced in preeclamptic women when compared with those with obstructive sleep apnea alone. Several investigators have suggested that obstructive sleep apnea (OSA) may be common in pregnant women despite the presence of intrinsic mechanisms that seem to be geared towards preventing sleep apnea. However, the exact incidence and prevalence of sleep apnea in pregnant women is uncertain. In addition, it is unclear if criteria that are used to define sleep apnea in the general population should be applied to pregnant women. Further investigations are needed to determine if lower thresholds for management of OSA should be used in pregnant women to prevent harm to the fetus. SUMMARY: In conclusion, sleep disturbances are common during pregnancy though the full extent of this relation remains undefined. Large, multi-center, prospective studies are needed for better understanding.

 

 

Obstet Gynecol. 1989 Sep;74(3 Pt 2):453-5.

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Precipitation of obstructive sleep apnea during pregnancy.

Kowall J, Clark G, Nino-Murcia G, Powell N.

Sleep Disorders Clinic,
Stanford University, California.

A case of severe obstructive sleep apnea developing during pregnancy is reported. A 27-year-old primigravida was well until the sixth month of pregnancy, when she developed loud snoring and excessive daytime sleepiness. Polysomnography was performed at 36 weeks' gestation and revealed severe obstructive sleep apnea. The patient was treated successfully during pregnancy with nasal continuous positive airway pressure, but continued to suffer from moderate obstructive sleep apnea after delivery. This case suggests that sleep apnea may be either precipitated or exacerbated during pregnancy.

 

 

South Med J. 1998 Aug;91(8):761-2.

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Obstructive sleep apnea during pregnancy resulting in pulmonary hypertension.

Lewis DF, Chesson AL, Edwards MS, Weeks JW, Adair CD.

Department of Obstetrics and Gynecology,
Louisiana State University Medical Center, Shreveport 71130-3932, USA.

Obesity is known to increase maternal morbidity and mortality. We describe a case of obstructive sleep apnea due to obesity and discuss our treatment of the resulting pulmonary hypertension. A patient was transferred to our hospital at 29 weeks' gestation with severe anasarca and more than a 100-pound weight gain during pregnancy. Pulmonary hypertension due to obstructive sleep apnea was diagnosed. The patient was treated with nasal continuous positive airway pressure (CPAP) during sleep and remained in the hospital the remainder of her pregnancy. She had a massive spontaneous diuresis during her hospital stay and lost more than 100 pounds. She was delivered at term via cesarean section because of transverse lie. Preoperative hemodynamic monitoring confirmed the diagnosis of pulmonary hypertension. This represents the first case in the literature of obstructive sleep apnea leading to pulmonary hypertension in pregnancy. This patient responded well to nasal CPAP as evident by the massive diuresis and good maternal outcome.

 

 

Rev Neurol (Paris). 2003 Nov;159(11 Suppl):6S91-4.

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[Respiratory disorders during sleep in the pregnant woman]

[Article in French]

Meurice JC, Paquereau J, Neau JP, Pourrat O, Pierre F.

Service de Pneumologie, CHU de Poitiers,
Poitiers. meurice@chui-poitiers.fr

Sleep disordered breathing can occur during pregnancy due to the development of hormonal changes and respiratory function abnormalities that perturb patency of the upper airways. Habitual snoring has been described in 25 p. 100 of the women during the third trimester of pregnancy. The incidence of sleep apnea hypopnea syndrome is unknown due to the lack of longitudinal epidemiological data, and results of the main studies are in favor of upper airway resistance syndrome. However, these sleep-related breathing disorders are more frequently associated with maternal and fetal complications such as maternal hypertension, pre eclampsia and intrauterine growth restriction. This article points out the importance of such associations because of the efficacy of continuous positive airway pressure on the regression of these nocturnal respiratory and vascular complications.


 

Harefuah. 2005 Feb;144(2):107-11, 150.

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[Maternal obesity as a risk factor for complications in pregnancy, labor and pregnancy outcomes]

[Article in Hebrew]

Raichel L, Sheiner E.

Department of Obstetrics and Gynecology, Faculty of Health Sciences,
Soroka University Medical Center, Ben Gurion University of the Negev, Beer Sheva, Israel.

Obesity is a worldwide growing epidemic. The negative influence of obesity is huge and considered to be one of the major contributors to health problems in the western world. There is a significant association between obesity and diabetes mellitus, ischemic heart disease, some cancers and syndromes of sleep apnea. Furthermore, obesity was described to have a negative influence on fertility, pregnancy, labor and pregnancy outcomes. It was also discovered that obesity was significantly associated with gestational hypertension, preeclampsia, gestational diabetes mellitus and complications in cesarean delivery and anesthesia. This review aims to present updates on the relationship between obesity and pregnancy and labor outcomes, emphasizing the significance of obesity as a risk factor for adverse pregnancy outcome.


 

Sleep. 2004 Nov 1;27(7):1405-17.

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Sleep disorders during pregnancy.

Pien GW, Schwab RJ.

Divisions of Sleep Medicine and Pulmonary, Allergy & Critical Care, Department of Medicine, University of Pennsylvania School of Medicine, Philadelphia, PA 19104, USA. gpien@mail.med.upenn.edu

This paper reviews the topic of sleep disorders in pregnant women. We describe changes in sleep architecture and sleep pattern during pregnancy, discuss the impact of the physical and biochemical changes of pregnancy on sleep in pregnant women and examine whether maternal-fetal outcomes may be adversely affected in women with disordered sleep. The literature on common sleep disorders affecting pregnant women, including insomnia, sleep-disordered breathing and restless legs syndrome, is reviewed and recommendations are made for the management of these disorders during pregnancy.

 

 

Acta Obstet Gynecol Scand. 2004 Feb;83(2):159-64.

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Arterial oxygen tension during sleep in the third trimester of pregnancy.

Prodromakis E, Trakada G, Tsapanos V, Spiropoulos K.

Division of Pulmonology, Laboratory of Sleep and the Department of Obstetrics and Gynecology, University of
Patras Medical School, Patras, Greece.

BACKGROUND: Changes during pregnancy include reduced functional residual capacity (FRC) and residual volume (RV), increased alveolar difference for oxygen and, in the supine position, reduced cardiac output. In conjunction with sleep-related disturbances, these changes could lead to maternal oxygen desaturation during sleep. OBJECTIVES: Because of conflicting data from respiratory sleep studies in pregnancy, we performed complete polysomnography on 21 pregnant women at the 36th week of gestation and again postpartum. We also measured the partial pressure of oxygen in the arterial blood (PaO2) in the supine and sitting positions. METHODS: We tested 21 healthy pregnant women at the 36th week of gestation. Arterial samples were taken in the sitting position. Complete polysomnography was performed in all of the pregnant women. Before the polysomnography arterial samples were taken in the supine and sitting positions and then every 2 h until termination of the study. RESULTS: We did not find any correlation between SaO2/PaO2 levels and apnea, hypopnea or percent of rapid eye movement (REM) sleep. The frequency of apnea and hypopnea was significantly lower during pregnancy (5.81 +/- 2.1 apneas or hypopneas per hour of sleep) than postpartum (12.1 +/- 2.7 apneas or hypopneas per hour of sleep) (p < 0.001), which may be due to the raised level of progesterone. The PaO2 levels in the supine position were significantly lower than in the sitting position at 36 weeks of gestation (p < 0.001). No differences were found between PaO2 levels in the sitting and supine positions postpartum (p < 0.5). CONCLUSIONS: According to our results we conclude that 1) the frequency of apnea and hypopnea in pregnancy was significantly lower than postpartum, and 2) a significant difference in PaO2 levels in the sitting and supine positions was observed at 36 weeks of gestation.

 

 

Sleep Med. 2004 Jan;5(1):43-51.

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Pregnancy, sleep disordered breathing and treatment with nasal continuous positive airway pressure.

Guilleminault C, Kreutzer M, Chang JL.

Stanford University Sleep Disorders Clinic, 401 Quarry Road, Suite 3301, Stanford, CA 94305, USA. cguil@leland.stanford.edu

OBJECTIVE: To investigate the tolerance, compliance and problems associated with usage of nasal continuous positive airway pressure (CPAP) by pregnant women with sleep disordered breathing (SDB). PATIENTS AND METHOD: Twelve pregnant women diagnosed with SDB received polysomnography (PSG) at entry, CPAP titration, repeat PSG at 6 months gestation (GA) and home monitoring of cardio-respiratory variables at 8 months GA. Compliance was verified by the pressure at the mask. Results from the Epworth sleepiness scale, fatigue scale and visual analogue scales (VAS) for sleepiness, fatigue, and snoring were compared over time. RESULTS: All of the subjects had full term pregnancies and healthy infants. Nightly compliance was at least 4 h initially and 6.5 h at 6 months GA. Nasal CPAP significantly improved all scales compared to entry. VAS scores remained lower at 6 months GA compared to entry. Re-adjustment of CPAP pressure was needed in six subjects at 6 months GA. CONCLUSION: Nasal CPAP is a safe and effective treatment of SDB during pregnancy.

 

 

 

Curr Treat Options Neurol. 2004 Jul;6(4):319-330.

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Sleep Problems Across the Life Cycle in Women.

Moline M, Broch L, Zak R.

Eisai Incorporated, Glenpointe Centre West,
500 Frank W. Burr Boulevard, Teaneck, NJ 07666, USA. margaret_moline@eisai.com

Across the life cycle of women, the quality and quantity of sleep can be markedly impacted by internal (eg, hormonal changes and vasomotor symptoms) and external (financial and child-care responsibilities; marital issue) factors. This paper will outline some of the major phases of the life cycle in women that have been associated with sleep problems. The main messages from this paper include 1) that very little systematic, large-scale research has been performed in virtually every area reviewed; and 2) once identified, the sleep problem is generally best addressed by the standard therapeutic approach, except in the case of pregnant and lactating women in which concern for the fetus and child must be considered in the treatment decision. This paper is organized into sections that address sleep problems associated with the menstrual cycle, pregnancy, postpartum, and perimenopause. Anecdotal reports recommend treatment that addresses the specific physical discomfort experienced by the woman (eg, analgesics for premenstrual pain, pregnancy pillows for backache, and hormone replacement therapy for hot flashes). The importance of developing standard treatment recommendations is stressed because the development of chronic insomnia has been linked to precipitating events. In addition, primary sleep disorders (eg, sleep apnea or restless legs syndrome) have been shown to increase during pregnancy and menopause, but treatment recommendations may be contraindicated or are not specific for women.



Clin Chest Med. 2004 Jun;25(2):257-68.

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Gender differences in sleep and sleep-disordered breathing.

Collop NA, Adkins D, Phillips BA.

Division of Pulmonary and Critical Care Medicine, Johns Hopkins University, 1830 East Monument Street, Room 555,
Baltimore, MD 21205, USA. ncollop@jhmi.edu

Sleep and sleep disorders are different in several important ways between men and women. Because of pregnancy and menopause, women experience changes in sleep that may present as clinical problems. In clinical populations, women are more likely to present with insomnia than are men, although their sleep may be better preserved. The presentation of sleep apnea in women is distinct from that of men and is less likely to include a "classic" history of witnessed ap-nea or heavy snoring. More likely it presents with nonspecific symptoms, such as fatigue or mood disturbance. There are little data on the effects of different treatments for OSA between men and women. OHS is a syndrome that may be as common in women as in men. The role of hormones in its pathophysiology is not well-defined.

 

 

Eur Respir J. 2003 Jul;22(1):161-72.

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Sleep-disordered breathing and hormones.

Saaresranta T, Polo O.

Dept of Pulmonary Diseases,
Turku University Central Hospital, University of Turku, Turku, Finland. tarja.saaresranta@tyks.fi

Sleep-disordered breathing (SDB) is not only a problem of the upper airway but is a systemic condition with endocrine and metabolic interactions. The accumulating body of evidence shows that SDB induces changes in the serum levels or secretory patterns of several hormones. Conversely, various endocrine disorders and hormone therapies may induce, exacerbate or alleviate SDB. Much of the understanding of the interactions between hormones and sleep-disordered breathing derive from intervention studies with nasal continuous positive airway pressure therapy. Better understanding of hormones and breathing may open new perspectives in developing strategies to prevent, alleviate or cure sleep-disordered breathing and its systemic consequences.


 

Sleep Med Rev. 2003 Apr;7(2):155-77.

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Sleep in women across the life cycle from adulthood through menopause.

Moline ML, Broch L, Zak R, Gross V.

Department of Psychiatry, Weill Medical College of Cornell University, New York Presbyterian Hospital, 21 Bloomingdale Road, White Plains, New York, 10605, USA.

Studies of sleep across the life cycle in women have utilized both survey and polysomnographic techniques, but have tended to be of small sample size with diverse methodology. As a result, definitive conclusions about the impact of the menstrual cycle and use of oral contraceptives on sleep parameters cannot yet be made. Sleep disruption during pregnancy and postpartum is nearly universal, but effective and practical countermeasures are still needed. Longitudinal studies of sleep in the postpartum period are also lacking. Menopause is associated with insomnia due to several factors including hot flashes, mood disorders and increased sleep-disordered breathing. The use of hormone replacement therapy to treat sleep and other variables is an active area of investigation. In summary, much research is required to fully elucidate the impact of the life cycle on sleep parameters in women.

 

Med Hypotheses. 1989 Sep;30(1):51-4.

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Obstructive sleep apnea (OSA)-implications in maternal-fetal medicine. A hypothesis.

Schoenfeld A, Ovadia Y, Neri A, Freedman S.

Department of Obstetrics and Gynecology,
Beilinson Medical Center Petah Tikva, Israel.

During the past fifteen years the obstructive sleep apnea syndrome (OSAS) has become widely recognized as a quite common disorder with a wide range of serious clinical complications (1, 2, 3). This syndrome arises as a result of sleep related changes in upper airway muscle function and progressive narrowing of the oropharyngeal lumen. The resulting hypoxia (or asphyxia) leads to an arousal response that terminates the initial obstructive event. The exact incidence and prevalence of OSAS is currently unknown. Lavie (4) concluded its prevalence to be 1.26 percent. Others (5, 6) found that the prevalence of heavy regular snoring (taken as an index of at least a partially obstructed airway) increased with age including 40 percent of women and 50 percent of men over 60 years of age. Polygraphically documented OSAS showed incidence of 0.99 percent in an unselected population. Postmenopausal women have frequent episodes. of OSA in contrast with their premenopausal counterparts, who very rarely have any apnea. Since we could not find in the literature any documented OSAS studies in pregnancy, we would like to base our hypothesis on our previously published clinical observations and our recent findings. In the present paper we would like to suggest that pathophysiologic changes of OSAS prolonged throughout many weeks of pregnancy may have an adverse effect upon the feto-placental unit.

 

 

Am J Hypertens. 2001 Nov;14(11 Pt 1):1090-5.

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Hemodynamic responses to obstructive respiratory events during sleep are augmented in women with preeclampsia.

Edwards N, Blyton DM, Kirjavainen TT, Sullivan CE.

Department of Medicine, The University of
Sydney, Australia. ne@blackburn.med.usyd.au

Preeclampsia is the most common disease of pregnancy, occurring in up to 10% of the pregnant population. The cause of the disease is as yet undetermined; however, most of the clinical effects are commonly attributed to damage to the endothelial layer, leading to increased pressor activity of all the maternal blood vessels. Therefore, we suspected that if obstructive sleep apnea (OSA) coexisted with preeclampsia in pregnancy, the hemodynamic effects of the OSA would be markedly potentiated. To test this hypothesis, we performed full sleep studies and overnight beat-to-beat blood pressure (BP) monitoring. The control patient group included 10 pregnant women with OSA and no evidence of hypertensive disease either before or during their current pregnancy. The test group included 10 women with preeclampsia and coexisting OSA. The pressor responses to obstructive respiratory events during sleep were enhanced in preeclamptic patients compared with control OSA patients (21+/-2/12+/-1 mm Hg and 38+/-5/25+/-4 mm Hg above baseline in control OSA and preeclamptic OSA patients, respectively, P = .005/.005). In contrast, there was no difference in heart rate responses between the two groups of subjects (34+/-5 beats/min and 49+/-13 beats/min above baseline in control and preeclamptic patient groups, respectively, P = .326). We suggest that the augmented pressor responses in preeclamptic women occur as a result of maternal endothelial damage induced by the preeclampsia disease process. These findings may have important implications in the management of preeclamptic patients.

 

 

 

Am J Respir Crit Care Med. 2003 Jan 15;167(2):137-40.

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The upper airway in pregnancy and pre-eclampsia.

Izci B, Riha RL, Martin SE, Vennelle M, Liston WA, Dundas KC, Calder AA, Douglas NJ.

Edinburgh Sleep Centre and Department of Reproductive and Developmental Sciences, University of Edinburgh, Scotland, United Kingdom.

Snoring is common in pregnancy, and snoring pregnant women have increased rates of pre-eclampsia. Patients with pre-eclampsia show upper airway narrowing during sleep. The present study aimed to compare upper airway dimensions in pregnant and nonpregnant women and in patients with pre-eclampsia. A total of 50 women in the third trimester of pregnancy and 37 women with pre-eclampsia were recruited consecutively from the antenatal service and matched with 50 nonpregnant women. Upper airway dimensions were measured using acoustic reflection. Comparisons were made by analysis of variance and Student-Newman-Keuls tests. Snoring was reported by 14% of nonpregnant women, 28% of pregnant women, and 75% of pre-eclamptic women (p < 0.001). When seated, pregnant women had wider upper airways than nonpregnant women (p < 0.02), but there was no difference when supine. Oropharyngeal junction area in the seated position was less (p < 0.01) in the women with pre-eclampsia (mean +/- SD: 0.9 +/- 0.1 cm2) than either nonpregnant (1.1 +/- 0.1 cm2) or pregnant women (1.3 +/- 0.1 cm2). Supine oropharyngeal junction area was less in the women with pre-eclampsia than in the nonpregnant women (0.8 +/- 0.1 versus 1.0 +/- 0.1 cm2; p = 0.01) but similar in women with pre-eclampsia and pregnant women (0.9 +/- 0.1 cm2; p > 0.3). The study showed that women with pre-eclampsia have upper airway narrowing in both upright and supine postures. These changes could contribute to the upper airway resistance episodes during sleep in patients with pre-eclampsia, which may further increase their blood pressure.

 

 

 

J Hypertens. 2001 Aug;19(8):1437-44.

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The prevalence and clinical significance of nocturnal hypertension in pregnancy.

Brown MA, Davis GK, McHugh L.

Department of Medicine, St George Hospital, University of New South Wales, Kogarah, Sydney, Australia. mbrown@unsw.edu.au

OBJECTIVE: To determine (a) the prevalence of hypertension during sleep in pre-eclampsia and gestational hypertension, and (b) whether women with hypertension during sleep have worse pregnancy outcomes than hypertensive pregnant women with controlled (normal) blood pressure (BP) during sleep. DESIGN: Prospective double-blind cohort study. SETTING: Inpatients and outpatients managed in a day assessment unit (DAU) at St George Hospital,
Sydney, Australia. PARTICIPANTS: A total of 186 hypertensive pregnant women, 158 of whom had successful 24 h BP monitoring; 40% had proteinuric pre-eclampsia (PE), 43% gestational hypertension (GH) and 17% essential hypertension (EH). INTERVENTIONS: Blood pressure, 24 h non-invasive, monitoring (Spacelabs 90207) was undertaken successfully in 158 women with PE, GH or EH, whether or not they were receiving antihypertensives. Women and clinicians were blinded to results of these BP monitors. Sleep hypertension was defined as BP > 117/68 mmHg at 26-30 weeks or > 123/72 mmHg after 30 weeks gestation. MAIN OUTCOME MEASURES: Maternal and fetal outcomes were compared between women with and without sleep hypertension and the prevalence of sleep hypertension was determined. RESULTS: Sleep hypertension was present in 59%, more commonly in PE (79%) than GH/EH (45%), P < 0.0001. Sleep hypertensives also had higher routine sphygmomanometer BPs [137(10)/91(7) mmHg; mean(SD)] than women with normal sleep BP [130(12)/ 87(8) mmHg] P = 0.007, and higher awake ambulatory blood pressure monitoring (ABPM) BPs [137(8)/88(7) versus 127(7)/79(6) mmHg], P < 0.0001. Awake, but not sleep, average heart rate was lower in sleep hypertensives [85(11) versus 91 (10) beats per minute, bpm], P = 0.002. Sleep hypertensives had a significantly greater frequency of renal insufficiency, liver dysfunction, thrombocytopenia and episodes of (awake) severe hypertension (P < 0.05), as well as lower birth weight babies [2715 (808) versus 3224(598) g, P < 0.0001]. CONCLUSIONS: Hypertension during sleep is a common finding in women with hypertensive disorders of pregnancy, particularly pre-eclampsia. These women also have higher awake BPs and a greater frequency of adverse maternal and fetal outcomes. These findings are largely explained by the greater likelihood of pre-eclamptics having sleep hypertension.

 

 

 

Eur Respir J. 2001 Oct;18(4):672-6.

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Inspiratory flow limitation during sleep in pre-eclampsia: comparison with normal pregnant and nonpregnant women.

Connolly G, Razak AR, Hayanga A, Russell A, McKenna P, McNicholas WT.

Depts of Obstetrics and Gynaecology,
Rotunda Hospital, Dublin, Ireland.

Self-reported snoring is common in pregnancy, particularly in females with pre-eclampsia. The prevalence of inspiratory flow limitation during sleep in preeclamptic females was objectively assessed and compared with normal pregnant and nonpregnant females. Fifteen females with pre-eclampsia were compared to 15 females from each of the three trimesters of pregnancy, as well as to 15 matched nonpregnant control females (total study population, 75 subjects). All subjects had overnight monitoring of respiration, oxygen saturation, and blood pressure (BP). No group had evidence of a clinically significant sleep apnoea syndrome, but patients with pre-eclampsia spent substantially more time (31+/-8.4% of sleep period time, mean+/-SD) with evidence of inspiratory flow limitation compared to 15.5+/-2.3% in third trimester subjects and <5% in the other three groups (p=0.001). In the majority of preeclamptics, the pattern of flow limitation was of prolonged episodes lasting several minutes without associated oxygen desaturation. As expected, systolic and diastolic BPs were significantly higher in the pre-eclamptic group (p<0.001), but all groups showed a significant fall (p< or =0.05) in BP during sleep. Inspiratory flow limitation is common during sleep in patients with pre-eclampsia, which may have implications for the pathophysiology and treatment of this disorder.

 

 

Chest. 2001 Nov;120(5):1448-54.

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Sleep-related disordered breathing during pregnancy in obese women.

Maasilta P, Bachour A, Teramo K, Polo O, Laitinen LA.

Department of Pulmonary Medicine,
Helsinki University Hospital, Helsinki, Finland. paula.maasilta@helsinki.fi

STUDY OBJECTIVES: This study was designed to evaluate sleep-related disordered breathing in obese women during pregnancy. Obesity is known to predispose to sleep-related breathing disorders. During pregnancy, obese mothers gain additional weight, but other mechanisms may counteract this effect. DESIGN: A case-control study to compare sleep-related breathing in obese pregnant women (mean prepregnancy body mass index [BMI] > 30 kg/m(2)) with pregnant women of normal weight (mean BMI, 20 to 25 kg/m(2)). SETTING: University teaching hospital with a sleep laboratory. PARTICIPANTS: We recruited 11 obese women (BMI, 34 kg/m(2); mean age 31 years) and 11 control women (BMI, 23 kg/m(2); mean age 32 years). INTERVENTIONS: Overnight polysomnography was performed during early (after 12 weeks) and late (after 30 weeks) pregnancy. MEASUREMENTS AND RESULTS: During pregnancy, obese mothers gained 13 kg and control women gained 16 kg. Sleep characteristics did not differ between the groups. During late pregnancy, the women in both groups slept more poorly and slept in supine position less. During early pregnancy, their apnea-hypopnea indexes (1.7 events per hour vs 0.2 events per hour; p < 0.05), 4% oxygen desaturations (5.3 events per hour vs 0.3 events per hour; p < 0.005), and snoring times (32% vs 1%, p < 0.001) differed significantly. These differences between the groups persisted in the second polysomnography, with snoring time further increasing in the obese. Preeclampsia and mild obstructive sleep apnea were diagnosed in one obese mother. One obese mother delivered a baby showing growth retardation (weight - 3 SD). CONCLUSIONS: We have shown significantly more sleep-related disordered breathing occurring in obese mothers than in subjects of